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Peritonitis, or inflammation of the peritoneum, is a serious concern warranting careful evaluation
Primary peritonitis (spontaneous bacterial peritonitis, SBP) usually in people with ascites due to liver disease or nephrotic syndrome.
Bacteria can enter ascites fluid through bowel or the fallopian tubes. Portal bacteremia can increase due to impaired function of liver reticuloendothelial cells with cirrhosis.
The predominant pathogens include
Secondary peritonitis can follow:
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Causative organisms can include:
Peritoneal dialysis is often caused by
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Tertiary peritonitis is a persistent abdominal infection leading to multiple organ failure, despite intensive surgical and antibiotic intervention. It represents severe immune and endocrine dysfunction, and has a mortality rate over 50%. Common pathogens, potentially a symptom rather than the cause, include:
Peritonitis can result in:
Primary
Patients are usually febrile and uncomfortable, preferring to lie quietly.
Other findings include:
Febrile patients with ascites should undergo paracentisis, with culture media innoculation at the bedside. Leukocyte count and gram stain can be helpful.
Mixed flora or anaerobes can follow bowel leakage.
Antibiotic penetration of the peritoneum is excellent. Piperacillin-tazobactam or ampicillin-sulbactam should be considered emperically.
Prophylactic norfloxacin or TMP-SMX may be helpful in patients with cirrhosis and ascites.
Peritonitis resulting from peritoneal dialysis can be treated by IV or intraperitoneal antibiotics, administered by the dialysis catheter. Vancomycin plus a cephalosporin should be used. The catheter need not always be removed, but should be with yeast, Pseudomonas, or polymicrobial infections.
created: DLP, Aug 09
authors: DLP, Aug 09
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